GC−MS analysis of 786-O and isogenic SETD2-deficient 38E/38F

SETD2 loss-of-function promotes renal cancer branched evolution through replication stress and impaired DNA repair

Reconstructed GC-MS chromatogram of the silylated total lipid extract.

ASG Analytik Service AG - GC x GC - MS

Simple and sensitive GC/MS-method for the quantification of urinary phenol, o- and m-cresol and ethylphenols as biomarkers of exposure to industrial solvents - ScienceDirect

PDF) Loss of SETD2 Induces a Metabolic Switch in Renal Cell Carcinoma Cell Lines toward Enhanced Oxidative Phosphorylation

The GC-MS fatty acid profile of refined olive oil: Palmitic

SETD2 loss-of-function promotes renal cancer branched evolution through replication stress and impaired DNA repair

Frontiers Histone methyltransferase SETD2: An epigenetic driver in clear cell renal cell carcinoma

Integrated Genomic and Proteomic Analyses Reveal Novel Mechanisms of the Methyltransferase SETD2 in Renal Cell Carcinoma Development - ScienceDirect

Evaluation of methylations and external/internal standard quantification of lipids using gas chromatography-mass spectrometry - Analytical Methods (RSC Publishing)

PDF) Loss of SETD2 Induces a Metabolic Switch in Renal Cell Carcinoma Cell Lines toward Enhanced Oxidative Phosphorylation

SETD2 loss-of-function promotes renal cancer branched evolution through replication stress and impaired DNA repair